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Recent press releases
Disruption of brain-blood barrier might influence progression of Alzheimer’s
29 September 2015
The team of Roosmarijn Vandenbroucke in the Claude Libert Group (VIB/UGent) combined their knowledge and expertise related to inflammation with the expertise in Alzheimer’s disease present in the Bart De Strooper Group (VIB/KU Leuven).
New Diversity for Lager Beers
25 September 2015
Unlike ales, lager beers differ little in flavor. But now, by creating new crosses among the relevant yeasts, Kevin Verstrepen, PhD, Stijn Mertens, and their collaborators at VIB/KU Leuven have opened up new horizons of taste.
Alzheimer protein APP also involved in Fragile X Syndrome
24 September 2015
Emanuela Pasciuto and Claudia Bagni have discovered a novel role for APP, the protein involved in Alzheimer disease. APP plays a crucial role in the development of the Fragile X Syndrome, the most common cause of inherited intellectual disability.
Safety at VIB, what does it mean?
22 September 2015
Safety has always been and will always be important in our labs. Everyone has his or her part of responsibility. Over the years, the organization of safety within VIB has developed and become more explicit.
Meet our local VIB safety coordinators
22 September 2015
Take a look at what our local VIB safety coordinators have to say about their efforts to keep VIB a safe place to work.
Interplay between universities and VIB
22 September 2015
VIB forms a joint-venture structure with its partner universities. Our departments and research groups are both vib and university and are physically integrated in the buildings of the university.
Careful! It may be infectious
21 September 2015
The list of organisms and materials we work with and which may be infectious is steadily growing. Therefore the risk of laboratory acquired infections is also growing. Where do the major risks lie?
Unexpected role for the IKK complex in protecting cells from death
18 September 2015
The team of Prof. Bertrand in the group of Prof. Vandenabeele, demonstrates that the IKK complex protects cells from death by inactivating RIPK1, thereby revealing an unexpected NF-kB-independent new role of the IKK complex.
Alzheimer’s-disease-related proteases, BACE1 and APH1B-y-secretase, control axonal guidance by regulating growth cone dynamics
14 September 2015
BACE1 is the major drug target for Alzheimer’s disease. Soraia Barão and Bart De Strooper (VIB/KU Leuven) now show that this protease is critically involved in axonal guidance processes in thalamic and hippocampal neurons.
Scientists learn how to predict plant size
11 September 2015
VIB and UGent scientists have developed a new method which allows them to predict the final size of a plant while it is still a seedling.
Growing up on a farm provides protection against asthma and allergies
3 September 2015
Researchers at VIB (a leading life sciences institute in Flanders, Belgium) and Ghent University have successfully established a causal relationship between exposure at a young age to so-called farm dust and protection against asthma and allergies.
Most cited article published in Cell Death & Disease is from the Peter Vandenabeele lab (VIB/UGent)
28 August 2015
The journal Cell Death & Disease gives an overview of their most cited papers. N°1 in Europe is one from the group of Peter Vandenabeele.
VIB n°1 in patent citations in survey the times - Most "innovative" universities
26 August 2015
In a survey done by The Times on most innovative universities VIB ranks 1st in patent citations. Johan Cardoen (managing director VIB): ‘This is a great result and a very nice proof that the VIB model works.’
Various avenues to counteract TNF - crucial to tackling acute inflammation
25 August 2015
The cytokine TNF plays and important role in acute inflammation and in a number of auto-immune conditions. TNF needs to pass on a signal in order to execute its role. This can be achieved by binding to a receptor.
New insights in pathological mechanism that causes dysfunctional synapses
20 August 2015
Genetic analysis of human patients has shown that mutations in genes involved in synaptic communication can drive neuropsychiatric and neurological diseases such as autism spectrum disorder and Alzheimer’s disease.
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